| 摘要 |
Hypercyanogenesis is diagnosed by injecting a human patient parenterally with hydroxocobalamine to produce cyanocobalamine, collecting the urine of the patient, decomposing the cyanocobalamine eliminated with the urine by photolysis, and determining the quantity of the thus-liberated hydrocyanic acid. The cyanide level following injection will vary, always initially increasing. In a normal patient, the cyanide level will rapidly thereafter fall to that of hypocyanosis; but in a patient afflicted with persistent hypercyanogenesis, the cyanide level will fall only relatively slowly toward its initial value. |
